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[1]阿尔茨海默病新机制——GABAA受体功能受损导致神经元过度兴奋,进行,
[2]病理性衰老过程中小鼠全脑神经/血管网络成像新方法,进行,
[3]阿尔茨海默病的新靶点——脑特异雌激素合成酶,进行,
[4]利用单细胞质谱技术研究阿尔茨海默病神经元代谢紊乱及机制,进行,
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